In addition to the limited research into antibiotics, we are finding some new ways to fight bacteria. Among these are bacteriophages (or just phages) which are viruses that target bacteria. They're a little harder to use because they have to be targeted to a specific kind of bacteria (which mean doctors need to know what type of bacteria a patient has), but the fact that antibiotics can attack a broad spectrum of bacteria has actually made it easier for bacteria to gain resistance to antibiotics.
The biggest problem hasn't been that evolution helps bacteria defeat antibiotics, it's that we've done a poor job keeping up our part of the fight. After discovering penicillin, Alexander Fleming told people that we'd have a problem with resistance if we weren't careful. But instead of listening, antibiotics have been over prescribed--often to patients who aren't sick because of bacteria (they do not work on viruses), or simply to farm animals to promote growth (which hasn't even been show to work).
Even where they're medically helpful, we could be doing more to at least slow the development of resistance. First, if we prescribed multiple antibiotics at the same time, it could wipe out more of the bacteria in the body, instead of leaving behind a handful, not enough to leave us sick, but enough to allow resistance to evolve. Second, one of the biggest problems (and this is harder to solve) is that patients don't always take their antibiotics as long as they're supposed to. They stop when they're feeling better, but before they've killed the bacteria they're targeting.
So if we're careful, the new antibiotics could have longer lives than the old ones (and penicillin is still occasionally useful). But, here's where it gets really cool, it's been shown (at least in the lab) that bacteria that have been treated with phages are then more susceptible to antibiotics, even if the bacteria have grown resistant to those antibiotics. In short, what we need to do is be really smart about how we use our drugs.
Another reason the future might not be all that bleak is that resistance can be lost. Carrying around and using the genes for resistance comes at a fitness cost for bacteria--one that is well rewarded if they encounter the antibiotic they're resistant to. But it's possible--again if we're very careful--that bacteria could lose resistance if an antibiotic isn't used for a while (a long while). We've seen something similar with drug resistant strains of malaria (malaria is a single celled parasite, but not a form of bacteria) where resistance has been reigned in because the anti-malarial hasn't been used. (That's more complicated than I can quickly describe, and the process will be harder for antibiotic resistance)
TLDR: Things don't look as bleak as they did even ten years ago, but we need to be very smart about how we use these drugs.